肝螺杆菌感染引起VDR-/-小鼠炎性肠病相关肠纤维化模型的建立及机制探讨
吴志浩1(), 曹舒扬2, 周正宇1()()
Establishment of an Intestinal Fibrosis Model Associated with Inflammatory Bowel Disease in VDR-/- Mice Induced by Helicobacter hepaticus Infection and Mechanism Exploration
WU Zhihao1(), CAO Shuyang2, ZHOU Zhengyu1()()

图2. H.hepaticus感染后各组小鼠的结肠长度
注：A为各组1只小鼠的代表性结肠长度对比照片；B为H.hepaticus感染导致结肠缩短，并且VDR缺陷加重了这一病变（**P<0.01，n=5）。

Figure 2. Colon length of mice in each group after H. hepaticus infection
Note：A， Comparison photos of representative colon lengths of one mouse from each group； B， H.hepaticus infection resulted in a significant reduction in colon length， and VDR deficiency further exacerbated this pathological effect（**P<0.01， n=5）.